Therapeutics: Phosphoinositide 3-kinase α (PI3Kα); phosphoinositide 3-kinase catalytic subunit α polypeptide (PI3KCA; p110α); mammalian target of rapamycin (mTO


Cancer

INDICATION: Cancer

Patient sample and mouse studies suggest inhibiting the PI3Kα/mTOR pathway could help treat venous malformations. Genetic sequencing identified activating mutations in PI3KCA in venous malformation lesions from

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